A great explanation of nutritional ketosis

My goal is to stay in a state of nutritional ketosis at all times.  To get to a state of nutritional ketosis, your body must become keto-adapted, hence the name of this blog.

I’ve tried my best to explain both in this blog and verbally to others what nutritional ketosis means, but I recently ran across a great blog article that explains it far better than I ever could.  The post is appropriate named, “What is nutritional ketosis” and I hope you enjoy reading it at least half as much as I did!

Fat and cholesterol

It’s a little humorous to watch some people recoil when they learn about all of the fat I eat.  It’s usually followed up with a question about my cholesterol, or with a comment about how my arteries are going to “clog up.”

But I don’t give anyone too hard of a time because that is exactly how I would have reacted a year ago, and prior to the hours and hours of research I’ve done on this topic.

Let me first tell you the books I’ve read, with a plea to anyone reading this to do your own research and reach your own conclusions.  Your health is too important to blindly believe anyone, including me.  Books I’ve read that cover this topic include:

“The Great Cholesterol Myth: Why Lowering Your Cholesterol Won’t Prevent Heart Disease,” by Johnny Bowden, Ph.D., C.N.S. and Stephen Sinatra, M.D., F.A.C.C.

“Cholesterol Clarity,” by Jimmy Moore, and Eric C. Westman, M.D.

“The Big Fat Surprise: Why Butter, Meat & Cheese Belong in a Healthy Diet,” by Nina Teicholz

“Grain Brain: The Surprise Truth About Wheat, Carbs, and Sugar – Your Brain’s Silent Killers,” by David Perlmutter, M.D.

I couldn’t possibly cover everything in these books, but I will outline some of the most interesting and compelling conclusions reached in them.

  1.  Our brain needs cholesterol to thrive.  It is a “critical brain nutrient essential for the function of neurons” according to Dr. Perlmutter. Cholesterol is an essential fuel for the neurons, which can’t generate significant cholesterol on their own.  They rely on delivery of cholesterol from the bloodstream by a specific carrier protein – LDL.  LDL stands for low-density lipoprotein, and Dr. Perlmutter asserts that there is “absolutely nothing bad about it.”  The fundamental role of LDL in the brain is to capture cholesterol and transport it to the neuron.
  2. When LDL becomes damaged and can no longer deliver cholesterol to the brain is when trouble starts.  What is the most common way it is damaged?  By glucose.  Dr. Perlmutter explains, “Sugar molecules attach themselves to LDL and change the molecule’s shape, rendering it less useful while increasing free radical production.”
  3. LDL has been labeled the “bad cholesterol” but it is what makes up the LDL that matters.  There are dense particles, and fluffy particles.  The fluffy, type A particles are harmless.  The dense particles (referred to as type B LDL) are not, and may be a risk factor for heart disease. It takes a particle test (not always widely available) to tell the difference.
  4. A better predictor of heart disease is the ratio of triglycerides to HDL. (Bowden and Sinatra).  The authors suggest a ratio of around 2.  Therefore, if your triglycerides are 100 mg/dL and your HDL is 50 mg/dL, your ratio is 2.  In “Cholesterol Clarity” the doctors interviewed there also suggest a ratio of total cholesterol to HDL as a guide to alert you to a problem.  If the total cholesterol to HDL ratio is 3, then there is no need for concern.  If the ratio is closer to 7, that indicates a problem.  The book also suggests keeping triglycerides below 150 mg/dL.
  5. All of the authors in each book concur that a high-fat (good fats), low-carb diet will improve HDL and triglyceride levels.
  6. Eating foods with cholesterol has a minimal effect, if any, on serum cholesterol.  (Teicholz)  You’ll find reference to several studies in “The Big Fat Surprise” where this finding was made.  When we do eat foods with high amounts of cholesterol, the body responds by lowering the amount of cholesterol naturally produced.

I think this is enough for one post, but as I re-read these books and others I’ll be sure to post more information that I hope others find helpful.

Setting PRs, battling food

Since returning from LOTOJA, our rides have been much shorter, and we’ve gone back to climbing when we can.  Each ride has resulted in a personal record (PR) for one or both of us on a portion of the ride.

I had been anxious to climb Mt. Baldy since returning from France, but training for LOTOJA had to come first.  We finally made it back to Mt. Baldy last weekend, and while it is still a very tough climb – 6.9% average grade with sustained 15% at the very top – it was much more fun to climb this time.  Both Jonathan and I set PRs from Mt. Baldy Village to the top, and Jonathan set some on the bottom part as well.

This past weekend (2 days ago) we were not planning to ride hard as I was not having a good day on the bike.  As an aside, it’s been months since I’ve had to say that…I guess I was due for a “bad day.”  But what I thought was a bad day actually wasn’t when I looked at my results.  I was riding harder than I realized and just didn’t feel great.

We climbed one of our favorite local hills – not too steep but enough to wake up your legs – and were returning home when we connected with a group that had a very strong rider in the front.  There were 2 times I looked down at my computer to see we were going 32 mph. And we were going this speed in the flats – not downhill!  It was the first time I’d been able to sustain anything close to that speed on a flat road so I was pretty excited.  Once the super strong rider pulled off, our speed hovered around 26 before another guy accelerated and we were back to 31 mph.  Thankfully our turn for home came up right before it was my turn to pull!

Food has been a challenge for me the past few weeks.  Despite fasting throughout the morning, my ketosis levels have been very low – .5 or less.  When I do eat, my stomach hurts or I feel bloated.  Normally this happens when I eat too many sugar alcohols so I’m trying to be very careful about that, but the past few days it hasn’t seemed to matter.  I feel like the person in the commercial whose food is hitting her in the face.

I am going to continue fasting in the morning, and trying to only take in fat in liquid form for most of the day, and then be very particular about the food I do eat in the late afternoon and evening.  I’m hoping to narrow down the problem foods so I can stop the stomach aches and bloating.  My energy levels have been pretty good (no highs followed by lows) but I do need to figure out why my stomach is upset or bloated so much of the time.

Last note – I’ve been reading some terrific books on cholesterol and how fat ISN’T connected to heart disease.  I plan to post on both topics as soon as I’ve finished the second book, “The Big Fat Surprise.”